Abstract
Rationale: Mood and gastrointestinal disorders are frequently observed in patients with anorexia nervosa, while the role of gut-brain axis has been recently highlighted in food intake and behavior regulation. In the activity-based anorexia (ABA) model, an increase of colonic paracellular permeability has been reported. Thus, we aimed to evaluate the effects of glutamine (Gln), the preferential substrate for rapidly dividing cells, on intestinal barrier function in ABA mice.
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