Abstract
Physical exercise brings an increased demand for substrates to be metabolized in working muscle. Part of this requirement is met by glucose, circulating in the blood, released from an increase in hepatic glycogenolysis and through the use of glycogen in the muscle.
Highlights
Physical exercise brings an increased demand for substrates to be metabolized in working muscle
This study proposed that the glucose minimum value would be more affected by the initial high intensity bout of exercise
The heart rates achieved by the swimmers during the fastest swim (185 ± 11 beat/min - male; 182 ± 8 beat/min - female) and blood lactates recorded (11.9 ± 2.7 mmol/l - male; 10.8 ± 2.14 mmol/l - female) indicate the very high intensity of these swims
Summary
Physical exercise brings an increased demand for substrates to be metabolized in working muscle. Part of this requirement is met by glucose, circulating in the blood, released from an increase in hepatic glycogenolysis and through the use of glycogen in the muscle [1]. The level of Glucose-6-Phosphate (G-6-P) in the muscle cell exerts a controlling action over Hexokinase which moderates the transfer of glucose by the GLUT4 carriers across the cell membrane [4]. Increase in glycogen use can raise the G-6-P level that prevents further influx of glucose into that cell. Different cells may express different requirements (1 increasing glucose uptake, the other blocking glucose uptake) depending upon the demands being made on the muscle at that time [5]. The rise in glucose use when looking at oxidation rates and focused on the crossover point between free fatty acid and glucose metabolism was observed to be at 65% of relative intensity [6]
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