Abstract
“IT has been postulated that a gradual accumulation of ketone bodies in the system might be responsible for the onset of hyperglycemia and that the rise of blood sugar might be due not only to overproduction or nonutilization of glucose but, also, to a natural mechanism to combine with some toxic metabolites in the system.”1. It has been proposed that the condensation product of glucose and acetoacetate (furoate condensate) prevents alloxan diabetes even when the product is in very low concentration, and that this condensation product is formed in the urine of normal people and not in those of diabetic patients2. A new route of glucose utilization and ketolysis is suggested, therefore3.
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