Abstract
Background Hydrocephalus causes reactive astrocytosis and microgliosis throughout the brain but the global response of proinflammatory cytokines is not known. Thus we sought to characterize common inflammatory markers in a neonatal model of obstructive hydrocephalus. Our main hypothesis was that neuroinflammation would progress in neonatal hydrocephalus proportional to ventriculomegaly. In addition, the long-standing uncertainty about the possible global inflammatory effects of kaolin could also be addressed.
Highlights
Hydrocephalus causes reactive astrocytosis and microgliosis throughout the brain but the global response of proinflammatory cytokines is not known
53rd Annual Meeting of the Society for Research into Hydrocephalus and Spina Bifida Meeting abstracts - A single PDF containing all abstracts in this Supplement is available here.
Our main hypothesis was that neuroinflammation would progress in neonatal hydrocephalus proportional to ventriculomegaly
Summary
53rd Annual Meeting of the Society for Research into Hydrocephalus and Spina Bifida Meeting abstracts - A single PDF containing all abstracts in this Supplement is available here.
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