Abstract
Gliomatosis peritonei (GP) is an infrequent occurrence, exclusively associated with a mature or immature teratoma of the ovary. GP is defined as miliary implantation of glial tissues on the surface of the visceral or parietal peritoneum with secondary maturation into glial nodules of 1–10 mm. Robboy and Scully have suggested three possible sources of GP: (1) deposition of immature neural tissue with consequent maturation; (2) lymphogenous metastasis; and (3) mature glial cells extruded through a defect in the capsule of the primary tumor [1]. Surgery is the basic treatment for both mature and immature teratomas as well as for peritoneal gliomatosis [2]. In immature teratoma associated with GP, combined chemotherapy is recommended. Surgery and chemotherapy can give longer survival even in recurrent disease [3].
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