Abstract
A molecular marker for Gilbert Syndrome (GS) was recently identified (Bosma et al, NEJM 1995;333:1171-5). GS is associated with hyperbilirubinemia and decreased bilirubin UDP-glucuronosyltransferase activity (B-UGT) in adults. However, GS is usually diagnosed after puberty and the role of GS in the neonatal period is unknown. The current study tested the hypothesis that this marker (an extra TA in the TATAA box of the B-UGT gene, UGTIA, promoter) is associated with neonatal jaundice.
Sign-in/Register to access full text options 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
