Abstract
Gentamicin, a widely used antibiotic, is a well-known nephrotoxin responsible for acute nonoligouric kidney injury and tubular dysfunction. Among the rarest of these is Bartter-like syndrome, which reportedly occurs in adults and children as a toxic manifestation of gentamicin therapy with unclear pathophysiology. Subclinical hypermagnesiuria and hypercalciuria are detected immediately after administering gentamicin. However, the responsible target of gentamicin-associated tubular damage is unclear. This report presents a case of Bartter-like syndrome induced by gentamicin with delayed recovery of as long as 6 weeks and reviews the literature for possible mechanisms of this complication.
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