Abstract
Multiple myeloma (MM) is an incurable plasma cell malignancy that arises from two precursor states: Monoclonal Gammopathy of Undetermined Significance (MGUS) and Smoldering Multiple Myeloma (SMM). Some of those patients rapidly progress to MM, while others remain asymptomatic over their lifetime. However, the genetic and molecular profiles that underlie this heterogeneity in disease progression are not yet elucidated.
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