Abstract
Gene effects were estimated for powdery mildew disease reaction in mungbean to know gene action involved for powdery mildew resistance. Six generations (P 1, P 2, F 1, F 2, B 1 and B 2), of three crosses, PIP 3-85-2×TARM 18, AKM 8802×TARM 18 and PM 9339×TARM 18 were grown for recording disease incidence (DI), per cent disease index (PDI) and area under disease progress curve (AUDPC). Uniformity in the trend was observed for these three parameters. AUDPC values revealed differential rate of disease development on various genotypes of mungbean. Highest rate of disease development was observed on AKM 8802 (973.65) and lowest on TARM 18 (323.82). Both additive and dominance gene actions were found to be important in inheritance of powdery mildew resistance (all the three characters) including non-allelic interactions. Duplicate type of epistasis was detected for disease incidence and complementary for PDI and AUDPC in most of the crosses. The selected plants from early segregating generations may be intermated to accumulate resistance genes and to fix resistance at desired level. Due consideration should be given to quantify disease using AUDPC values.
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
