Abstract
Three mutants resistant to the fungicide benomyl and two resistant to dicarboximides were developed from a wild-type strain of Talaromyces flavus. Based on sensitivity of various isolates to benomyl concentrations, two levels of resistance were recognized. One isolate with low resistance (LR) was resistant to 5 μg/mL, but sensitive to 50 μg/mL benomyl; and two highly resistant (HR) isolates grew uninhibited at 50 μg/mL. Crosses between resistant isolates and sensitive wild types, as well as between different resistant isolates, showed that the two levels of benomyl resistance are conferred by different alleles at a single locus. The LR allele pleiotropically brings about slow growth. The dicarboximide-resistant isolates were little affected (mean effective dose (ED50) > 250 μg/mL) by iprodione, vinclozolin, dichlozolinate, or Co 6054. Crosses showed that the two dicarboximide-resistant isolates carry allelic mutations that can be characterized by the level of osmotic sensitivity pleiotropically conditioned by them. Dicarboximide resistance was not linked to benomyl resistance.
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