Abstract
Transforming growth factor belta1 (TGFβ1), tissue plasminogen activator (tPA) and angiotensin-converting enzyme (ACE) are known to be associated with normal tissue irradiation damage. Genetic variation within these genes has been identified as risk factor for this damage. We hypothesized that the following single nucleotide polymorphisms (SNPs) of TGFβ1 509C/T, tPA -7351 C/T and ACE I/D are associated with radiation induced thoracic toxicity (RITT) in patients with non-small cell lung cancer (NSCLC).
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