Abstract
The intensity of heat shock protein synthesis by splenocytes is assessed in mice of two strains with genetically different sensitivity to antiproliferative effect of alkylating effect of maphosphamide. The content andde novo production of heat shock proteins in resistant BALB/c mice are higher than in sensitive DBA/2 mice. Exposure to heat shock increases cell resistance to antiproliferative action of maphosphamide.
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