Abstract
A study of the suppressive factor of Saccharomyces cerevisiae and its effects on the transmission of cytoplasmically-inherited erythromycin resistance showed that loss of erythromycin resistance was contingent on suppressitivity, and that strains carrying the suppressive factor continuously segregated progeny cells that lacked resistance. It was suggested that suppressitivity is due to the presence, in suppressive strains, of rapidly-replicating abnormal mitochondrial DNA, and that loss of erythromycin resistance (coded for by normal mitochondrial DNA) is due to the replicative superiority of abnormal mitochondrial DNA.
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Schedule a callMore From: Canadian journal of genetics and cytology. Journal canadien de genetique et de cytologie
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