Abstract
Microglial cells have emerged as important players in the pathophysiology of neuropsychiatric diseases. To better understand the potential causal role of microglia in brain pathology and to identify microglia-related targets for treatment in the long-term, there is a critical need to elucidate how genetic risk for neuropsychiatric brain disorders is related to microglial function. By applying a combination of genetic and transcriptomic analyses on the same samples of a set of different donors, one can elucidate which gene is under influence of which genetic variant, by so-called quantitative trait loci (QTLs).
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