Abstract
Sapovirus was detected in 7 of 95 stool specimens from children with gastroenteritis of unknown etiology in Sydney, Australia, from August 2001 to August 2002 and from February 2004 to August 2004, by using reverse transcription–polymerase chain reaction. Sequence analysis of the N-terminal capsid region showed all human sapovirus genogroups.
Highlights
Sapovirus was detected in 7 of 95 stool specimens from children with gastroenteritis of unknown etiology in Sydney, Australia, from August 2001 to August 2002 and from February 2004 to August 2004, by using reverse transcription–polymerase chain reaction
In our previous study [10], an infant infected with NK24 (SaV genogroup GV) had a fever for 11 days and vomiting for 3 days, which was notably longer than the duration of symptoms in other infants infected with SaV GI and GII strains
One sequence belonged to genogroup GIV and had ≈99% nt identity with the SW278 sequence, which recently caused an outbreak of gastroenteritis in adults in Sweden in March 2004 [1]. Another sequence belonged to Conclusions Little is known about SaV infections in Australia
Summary
Sapovirus was detected in 7 of 95 stool specimens from children with gastroenteritis of unknown etiology in Sydney, Australia, from August 2001 to August 2002 and from February 2004 to August 2004, by using reverse transcription–polymerase chain reaction. Sequence analysis of the N-terminal capsid region showed all human sapovirus genogroups. SaV was detected in 7 of 95 stool specimens from children 9 months to 7 years of age with previously unknown causes of acute gastroenteritis. This represented a minimum prevalence of 4.1% (7 of 170 specimens). White blood cells were detected in the stool specimens of 3 children infected with SaV genogroups GII, GIV, and GV (strains Sydney4106, Sydney, and Sydney4402, respectively). These results suggest that some SaV genogroups could be more virulent than others, additional studies are needed
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