Abstract
Eosinophilic gastrointestinal diseases (EGID) represent a spectrum of disorders classically defined by eosinophil-predominant inflammation. Currently, there are no consensus diagnostic criteria for non-esophageal EGIDs. In contrast to the esophagus, eosinophils are resident cells in the gastrointestinal mucosa; therefore, establishing histologic thresholds indicative of pathology is more challenging. EGID are associated with Th2 inflammation. The master transcriptional regulators of Th1 and Th2 inflammation are T-bet and GATA-3, respectively.
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