Abstract
The year has seen further evidence that Helicobacter pylori increases release of the acid-stimulating peptide gastrin and suppresses expression of the inhibitory hormone somatostatin in the gastric mucosa. Measurements of acid secretion have shown that H. pylori increases the duodenal acid load, but that under other circumstances the same bacterium decreases acid secretion. It is important to understand these different responses to H. pylori because they lead to different disease outcomes. While some are considering the effect of H. pylori on acid, others are exploring the effect of acid on H. pylori. The bacteria migrate from the antrum to the corpus of the stomach when acid secretion is suppressed with omeprazole.
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