Abstract
A series of 101 young men comprising alcoholics, patients presenting with non-ulcer dyspepsia (NUD) and healthy controls was investigated for incidence of antibodies against gastric parietal cells, thyroglobulin and thyroid microsomes and for chronic gastritis as revealed by gastric biopsy. Alcohol is shown to be a cause of chronic gastritis. The incidence of parietal cell antibody (PCA) was unrelated to either the alcohol intake or to the incidence and severity of chronic gastritis. Subjects with PCA showed no increased incidence of thyroid antibodies. There is thus no evidence for the involvement of an autoimmune process in the genesis of alcohol-induced chronic gastritis. The incidence of PCA is significantly less in non-ulcer dyspeptics than in either control subjects or in the remainder of the series as a whole. The interpretation of this finding is not clear. Chronic gastritis is no more or less common among NUD patients than among healthy controls.
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