Abstract

Gastric outlet obstruction is a common entity described with numerous benign and malignant causes. Historically, benign causes include Peptic Ulcer disease (PUD); however recent cases have reported other causes including Eosinophilic Gastroenteritis (EGE), Brunner’s Gland Hyperplasia (BGH) and Pancreatic Heterotopia (PH). We present an unusual case of a 31 year old male patient with severe gastric outlet obstruction requiring surgical intervention caused by all three pathologies in unison – EGE, BGH and PH. The patient underwent surgical resection with distal gastrectomy and Billroth II reconstruction. Histology confirmed the three pathologies and post-operative recovery of the patient was uneventful.

Highlights

  • Gastric Outlet Obstruction (GOO) is a clinical and pathophysiological syndrome due to a mechanical obstruction

  • GOO is the clinical and pathological sequence of any disease process that produces a mechanical impediment to gastric emptying [1]

  • Rare pathologies as Eosinophilic Gastroenteritis (EGE), Brunner’s Gland Hyperplasia (BGH) and Pancreatic Heterotopia (PH) have independently manifested as a cause of GOO

Read more

Summary

Introduction

Gastric Outlet Obstruction (GOO) is a clinical and pathophysiological syndrome due to a mechanical obstruction. There are several causes listed for GOO including benign as well as malignant lesions. Majority of cases are attributable to malignancy including pancreatic adenocarcinoma and gastric cancer. Rare benign causes are Eosinophilic Gastroenteritis (EGE), Brunner’s Gland Hyperplasia (BGH) and Pancreatic Heterotopia (PH). EGE is a rare disease that has been reported as a cause of GOO [1]. BGH is a benign lesion that is mostly asymptomatic. Occasionally they can attain larger sizes and cause symptoms of obstruction [5, 6]. PH is a rare condition which is generally asymptomatic, but may present as a cause of GOO [8, 9]. We present an unusual case where all the three rare entities caused pyloric obstruction in unison

Case Report
Discussion
Conclusion
Full Text
Paper version not known

Talk to us

Join us for a 30 min session where you can share your feedback and ask us any queries you have

Schedule a call

Disclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.