Abstract
Hydrogen sulfide (H2S), produced endogenously in mammalian cardiovascular system, attenuates ischemia/reperfusion-induced myocardial injury. The protective mechanism involves in free radical scavenging, inflammation inhibition, mitochondria preservation, signal pathway activation and ATP-sensitive potassium channel opening. Key words: hydrogen sulfide; myocardial protection; ischemia/reperfusion injury
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Schedule a callMore From: International Journal of Anesthesiology and Resuscitation
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