Abstract
Morphologic evidence of invasion of the cardiac muscle was present in 9 out of 17 patients with histologic evidence of wide-spread systemic clostridial infection. The cardiac lesions consisted of foci of myonecrosis containing numerous organisms, myocardial gaseous cysts, and clumps of organisms within the lumina of cardiac vascular and lymphatic channels. There was no inflammatory response to the invading clostridial organisms. Each of the patients with clostridial myonecrosis of the heart had clostridial infection in a number of other organs. The portal of entry was a gastrointestinal or respiratorytract ulceration. It is probable that the incidence of systemic clostridial infections will rise with the increasing use of chemotherapeutic agents, steroids, and antibiotics.
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