Abstract
Exercises of constant workload (90 watt) have been carried out during normoxia or hyperoxia (FI O 2 = 0.45). It has been shown that, in spite of a significant dispersion in the values of O 2 deficit and O 2 debt calculated, these values are related to the increased blood lactate level which contributes to the marked acidosis observed in both conditions of oxygenation. Hyperoxia reduces lactate level as well as the O 2 debt. In addition to the significant increase in arterial [H +] and P CO 2 , exercise provokes a slight decrease P CO 2 . It is suggested that the significant variations of these humoral factors might contribute to the control of ventilation during exercise in both conditions of oxygenation.
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