Abstract
We have reported connexin43 (Cx43) remodeling is regulated by renin-angiotensin-aldosterone system, and is an important arrhythmogenic substrate. In the cardiovascular system, L- and T-type Ca2+ channels (LCC and TCC) are present. Re-expression of TCC plays a critical role in arrhythmogenesis. We investigated the effects of aldosterone (Ald) -induced re-expression of TCC on Cx43 remodeling in rat cultured cardiomyocytes. Culture was exposed to Ald for 24 h. The expression of TCC and Cx43 was assessed.
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