Abstract
Introduction: Fat accumulation in the liver is associated with metabolic syndrome; and through NASH, it could progress to hepatic cirrhosis and carcinoma. Since lipocalin-2 (LCN2) synthesis in the liver is influenced by inflammatory and metabolic processes, we aimed to examine LCN2 expression in two rat models of diet-induced fatty liver.
Sign-in/Register to access full text options 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
