Abstract
RNA viruses are the causative agents of severe diseases in vertebrates. Upon viral infection, various intracellular signaling pathways are induced within the infected cells. While most of the different cellular signaling responses are initiated as antiviral defense mechanisms to counteract invading pathogens, they may also be exploited by viruses to support their replication. Recently, PI3K has been added to the growing list of signaling factors and pathways that are activated upon viral infections and regulate the replication process. Here, the current knowledge on RNA virus-induced PI3K-regulated signaling processes and how the pathogens take advantage of these activities within the infected cells is summarized.
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