Abstract

Cerebral Thrombemboli with left ventricular origin are occasionally seen in patients with post-infarction left ventricular aneurysm or dilatativer cardiomyopathy of non-ischemic cause. Freedom of medical history of cardiac disease and corresponding symptoms may delay the proper diagnosis, particularly in patients with distinct neurological symptoms and normal findings in electrocardiogram or cerebral MRI. We report on a rare case of long standing neurological symptoms and late diagnosis of a left ventricular thrombus without clinical symptoms or medical history of cardiac disease. The patient underwent a thrombus extraction via left ventricular apical approach. He was discharged from hospital after an uneventful course in our clinic with remaining mild neurological symptoms that were partially recurrent under physiotherapy and logopedic therapy in the course of the following two years. An echocardiographical evaluation of cardiac function and exclusion of a cardiac source of emboli as a first line diagnostic tool may have led to an early diagnosis. Therefore, it should be implemented in the routine examination, independent of cardiac history or present cardiac symptoms.

Highlights

  • Cerebral Thrombemboli with left ventricular origin are occasionally seen in patients with post-infarction left ventricular aneurysm or dilatative cardiomyopathy of nonischemic cause [1,2]

  • We report on a rare case of long standing neurological symptoms and late diagnosis of a left ventricular thrombus without clinical symptoms or medical history of cardiac disease

  • It should be implemented in the routine examination, independent of cardiac history or present cardiac symptoms

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Summary

Introduction

Cerebral Thrombemboli with left ventricular origin are occasionally seen in patients with post-infarction left ventricular aneurysm or dilatative cardiomyopathy of nonischemic cause [1,2]. Most of these patients have a history of angina or heart failure symptoms that is under medical or surgical treatment including pharmacological regimen and interventional or surgical therapy. Complications resulting from cerebral thrombemboli include transient ischemic attacks (TIA), prolonged ischemic neurological deficits or a manifest apoplexy [3]. The associated symptoms vary from regional sensory impairment to complete loss of motor function, requiring immediate initiation of further diagnostic steps and therapy [4,5]

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