Abstract

Folding at upper crustal levels occurs by bending of beds and flexural slip between beds. As a fold's interlimb angle decreases, changes in bed thickness and limb rotation are accommodated by various mechanisms, depending on deformation conditions. In the elastico-frictional (EF) regime, cataclastic flow may be the dominant mechanism for fold tightening. The Canyon Range (CR) syncline, located in the Sevier belt of central Utah, shows this type of deformation. The fold involves three thick quartzite units, with slight lithological variations between them. Fold tightening took place in the EF regime (<2 km overburden) by cataclastic flow, involving collective movement on a distributed network of fractures and deformation zones (DZs) from the micro- to the outcrop-scale. In detail, the degree of cataclastic deformation varies significantly across the fold due to minor variations in initial bedding thickness, grain size, matrix composition, etc. A cooperative relationship exists across different scales, and the fracture networks result in a fracture shape fabric that is relatively homogeneous at the outcrop-scale. The initial outcrop scale fracture/DZ network geometry is a product of the growth and linking of micro-scale cataclasite zones, which in turn is controlled by primary lithological variations. Once a fracture network forms, the material behavior of the fractured rock is unlike that of the original rock, with sliding of fracture-bound blocks accomplishing ‘block-controlled’ cataclastic flow. Thus, initial lithological variations at the micro-scale largely control the final deformation behavior at the largest scale. During progressive fold tightening, additional factors regulate cataclastic flow, such as fracture/DZ reactivation or healing, during folding. Although initial lithological variations in different units may produce unique network geometries, each unit's behavior may also depend upon the behavior of adjacent units. In the CR syncline, during the initial stages of cataclastic flow, the inherent nature of each quartzite unit results in unit-specific fracture network geometries. As deformation progresses, unit-specific networks begin to interact with those in surrounding units, resulting in feedback mechanisms regulating the later stages of network development. Thus, the nature of cataclastic flow changes dramatically from the initial to the final stages of folding.

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