Abstract
Lymphotropic polyomavirus (LPV) was first isolated in 1979 from a B-lymphoblastoid cell line of an African green monkey by zur Hausen and Gissman (10). This virus has some characteristics common to human polyomavirus, such as virion morphology, the presence of a closed circular double-stranded molecule of DNA, and in vitro transforming activity (7), but it is antigenically distinct from simian virus 40, BK virus, and JC virus (1). In cell cultures, LPV has a highly restricted host range for both human and monkey B lymphoblasts (2, 3), and seroepidemiological studies revealed that, in addition to sera from monkeys, many human sera had strong reactions in the presence of LPV antigens (8).
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