Abstract
When fibrinolytic activity of arterial blood from arteriovenous shunts in patients on regular dialysis treatment was compared with arterial blood of age-matched subjects without renal disease no statistically significant difference was observed. It is suggested that this may be due either to the damaged kidneys continuing to produce plasminogen activator or to the smaller amount of activator produced by the diseased kidneys being compensated by a reduced removal of it in the lungs.
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