Abstract
Recently, decreased fibrinolytic activity (FA) in plasma of heterogenous groups of patients with chronic low-back pain (CLBP) has been demonstrated1,2). A decreased FA may lead to impaired removal of fibrin, deposited secondary to lesions and tissue damage of the lumbar spine. Fibrin is able to induce an inflammatory reaction(3), thus leading to pain and possibly fibrosis. This could explain why some individuals develop arachnoiditis, post-laminectomy syndrome or symptoms due to spondylosis and disc degeneration, while others do not.In this study we have investigated whether decreased FA could be demonstrated in a prospectively and consecutively collected population of patients with CLBP.
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