Abstract
Allergic asthma is an inflammatory disease that is associated with airway hyperreactivity (AHR). AHR is characterized by cough, dyspnea and an enhanced sputum production in response to many environmental stimuli such as cold air, cigarette smoke and ozone. Evidence has been provided that sensory neurons, especially the unmyelinated C-fibers, might contribute to AHR. Airway innervating C-fibers express transient receptor potential vanilloid 1 (TRPV1) and transient receptor potential ankyrin 1 (TRPA1).
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