Abstract
The long-chain fatty acid receptor FFA4 exerts beneficial effects on glucose homeostasis and insulin secretion and is considered a potential target for type 2 diabetes therapy. FFA4 is expressed in islets, but its precise mechanism of action remains unknown. Previous studies from our group suggest that FFA4 regulates somatostatin secretion by delta cells. The objective of this study was to test the hypothesis that FFA4 agonists indirectly stimulate insulin secretion via inhibition of somatostatin release.
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