Abstract
In acute rat experiments (thiopental sodium narcosis), it is established that introducing a 5 % glucose solution into the stomach lowers a frequency of heart rates (HR) and an amplitude of potentials of smooth muscles of the stomach and simultaneously raises doubtfully and shortly the potentials of sooth muscles (PSM) of the colon. Food additives used in the glutamate sodium, benzoate, tartrazine complex do not influence HR, but as before, reduces PSM of the stomach, and in the thick gut precise exciting or brake responses are marked. In the preliminarily developed heart insufficiency conditions, the glucose infusion into the stomach does not change HR. At the same time, the action of glucose, as well as of food additives, still results in reliable reactions of the stomach and the thick gut. Important is the present work information on the ability of glucose and food additives to modulate the heart activity estimated through the indicators of modification of electrocardiogram teeth. As appeared, the infusion of the specified irritants in the stomach of rats in control and heart insufficiency is accompanied by precise enough changes in such indicators as the RST duration, the amplitude and duration of the Т-wave, i. e. the prognostic attributes of the opportunity of occurrence of heart аrrhythmia, fibrillations.
Highlights
a 5 % glucose solution into the stomach lowers a frequency of heart rates
Food additives used in the glutamate sodium
the present work information on the ability of glucose and food additives to modulate the heart activity estimated through the indicators of modification
Summary
В задачу настоящей работы включено выявление изменений активности гладких мышц желудка, ободочной кишки и одновременно деятельности сердца по показателям ЭКГ после введения в желудок глюкозы на фоне сформированной сердечной недостаточности. 1. Эффект вводимой в желудок глюкозы в контрольных опытах (a) и после формирования хронической сердечной недостаточности (b). На фоне ХСН, как оказалось, введение на завершающем этапе в острых опытах данных препаратов регулярно приводит к понижению амплитуды ПГМ как желудка, так и ободочной кишки
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