Abstract

Bursitis is an inflammatory process that affects the joint capsule located between the bones and tendon fibers. In the normal state, the joint capsule is filled with synovial fluid the main function of which is to provide hydraulic lubrication for the normal sliding of the tendon along the bone in order to void damage to the tendon fibers. Depending on the location of the lesion of a particular anatomical region, there is bursitis of the shoulder joint (supracromial, subacromial, subdeltoid, subcoracoid forms), elbow joint, knee joint (prepatellar, suprapatellar, infrapatellar bursitis), the Achilles tendon bursitis, etc. Depending on the etiology of the lesion, specific bursitis caused by a certain type of pathogen and non-specific bursitis, most often of an autoimmune nature, are distinguished. Bursitis of the ankle joint can occur against the background of mechanical damage as a result of an injury, or it can develop against the background of gout or excessive load on the ankle joint. The main clinical manifestations of bursitis include pain, limited mobility, swelling, fever, and redness in the area of the affected joint. The tactics of treating ankle bursitis depends on its cause: antibiotic therapy is indicated for an infectious lesion; as for a traumatic injury, it is first necessary to ensure the immobilization of the affected limb, followed by the prescription of anti-inflammatory drugs; in autoimmune processes, doctors resort to the use of cor ticosteroids.

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