Abstract
Conversion of C14 into fatty acids of liver and carcass from acetate-2-C14, and into blood glucose and liver glycogen from DL-alanine-1-C14 were studied during and following a dynamic phase of obesity caused in rats by feeding of high caloric diet. (1) There were found to be no significant differences among normal rats and rats in dynamic or static (postdynamic) phases in cumulative expired C14O2 after injection of acetate-2-C14. (2) Conversion of C14 from acetate-2-C14 into fatty acids of the liver and carcass was increased in the dynamic phase and decreased in the static phase. (3) Incorporation of C14 from DL-alanine-I-C14 into blood glucose and liver glycogen was found to be increased in the dynamic and static phases of high-caloric fed rats. Conversion of C14 into fatty acids of liver and carcass from acetate-2-C14, and into blood glucose and liver glycogen from DL-alanine-1-C14 were studied during and following a dynamic phase of obesity caused in rats by feeding of high caloric diet. (1) There were found to be no significant differences among normal rats and rats in dynamic or static (postdynamic) phases in cumulative expired C14O2 after injection of acetate-2-C14. (2) Conversion of C14 from acetate-2-C14 into fatty acids of the liver and carcass was increased in the dynamic phase and decreased in the static phase. (3) Incorporation of C14 from DL-alanine-I-C14 into blood glucose and liver glycogen was found to be increased in the dynamic and static phases of high-caloric fed rats.
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