Abstract
A conceptual framework is presented that relates behavior pathology, behavior genetics, and the concept of mechanism-specific behavior to the study of alcohol preference of inbred mice as an approach to the understanding of human alcoholism. The programmatic work on alcohol preference of mice is reviewed. Genetic, enzymatic, nutritional, experiential, and stimulus variables are considered. Under appropriate circumstances, voluntary ingestion of sweetened alcohol by appropriate strains is shown to raise rate of mortality and to induce liver pathology similar to that found in some types or stages of human alcoholism.
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
