Abstract
Acetylcholinesterase (AChE) at neuromuscular junctions (nmj’s) is concentrated in the synaptic cleft. AChE consists of several molecular forms, some of which are specifically adherent to synaptic basal lamina (for review, see 20). Since all molecular forms of AChE have similar catalytic activity the major factors that determine the efficacy of AChE action in synaptic function involve the position of AChE molecules in the synaptic cleft, the cellular origin and regulation of AChE molecular forms, and the density of AChE. Various aspects concerning these factors have been studied recently in our laboratory and are reviewed in this chapter.
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