Abstract
Glasshouse experiments with a series of near-isogenic lines showed that the st gene, which reduces stipule area, and the tl gene, which converts tendrils to leaflets, considerably increased susceptibility to systemic infection with Peronospora viciae . Similar effects were observed with these pea lines when naturally infected in the field, indicating that direct infection of the apex is an important route for systemic infection and that its incidence is influenced by morphology of the apical primordia. In a growth room experiment at 25 °C where leaf necrosis was associated with inhibited sporulation, symptomless invasion of the stem tissues resulted in systemic infection, indicating that some apical infection may originate from leaf lesions under high temperature conditions.
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