Abstract
Trigeminal sensory neuropathy can be caused by a variety of conditions, including local, traumatic, iatrogenic, or systemic causes. Diagnosis and management remain a challenge for maxillofacial surgeons and neurologists. Therefore, a good clinical examination and objective tests and imaging are needed when diagnosing patients who present with facial numbness. We present a case with spontaneous episodes of facial paresthesia. He was diagnosed with hereditary neuropathy with liability to pressure palsies (HNPP), a rare condition that affects the peripheral nerves. Only a few case reports that describe involvement of the cranial nerves in patients with HNPP were found in the literature, and facial paresthesia has not been previously reported.
Highlights
According to the International Association for the study of Pain (IASP) terminology [1], paresthesia is defined as an abnormal sensation, whether spontaneous or provoked
A 25-year-old Afro-American male was referred to the Department of Oral and Maxillofacial Surgery by his family doctor because of recurrent episodes of facial paresthesia
Finding the cause of trigeminal sensory neuropathy can be a diagnostic challenge because it can be caused by a variety of disorders
Summary
According to the International Association for the study of Pain (IASP) terminology [1], paresthesia is defined as an abnormal sensation, whether spontaneous or provoked. The involvement of cranial nerves has seldom been described in case studies of HNPP The purpose of this case report is to review this rare presentation and to examine the process for differential diagnosis in a patient with spontaneous paresthesia in the distribution area of the trigeminal nerve. A 25-year-old Afro-American male was referred to the Department of Oral and Maxillofacial Surgery by his family doctor because of recurrent episodes of facial paresthesia. Electromyography (EMG) of the upper and lower limbs showed multifocal demyelinating anomalies with diminished sensory and motor conduction velocity In this regard, he occasionally encountered transient episodes of muscle weakness and hypoesthesia in the arms and legs, as mentioned earlier. Vitamin B supplements were interrupted without recurrence or exacerbation of symptoms
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