Abstract

Neurological damage following electrical burns is an often discussed complication. The spectrum of neurological sequelae includes cerebral injury, spinal cord lesions, peripheral sensory and motor neuropathies [1]. These sequelae may present early or may have a latent period lasting for several days prior to onset of the dysfunction. This case describes primary CNS injury with delayed development of myoclonus and athetosis in a patient who suffered high-tension electrical contact burns. This is an unusual complication and the probable mechanism is discussed.

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