Abstract
Earlier surveys of cerebal palsy characterized patients with extrapyramidal (choreoathetoid) cerebral palsy as having little cognitive impairment and few associated deficits. Anoxia, which has now replaced neonatal hyperbilirubinemia as a major cause of this type of cerebral palsy, predictably produces a diffuse type of brain damage. Evaluations of children with extrapyramidal cerebral palsy seen in a pediatric habilitation center revealed high incidences of mental retardation, epilepsy, and other associated deficits. These findings have implications for the management and habilitation of children with extrapyramidal cerebral palsy.
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