Abstract
Background: Small bowel resection (SBR) induces adaptation that is both structural (lengthened villi) and molecular (altered gene expression). The latter involves changes in nutrient, and especially lipid, processing. We previously demonstrated that intestinal extracellular matrix (ECM) can direct lineage commitment of endodermal progenitor cells toward intestinal epithelial precursors. Since ECM influences cellular gene expression, we hypothesized ECM critically affects not only …
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