Abstract
Primary anetoderma is a rare elastolytic disorder characterized by well-circumscribed flaccid, atrophic macules and patches caused by focal loss of elastic fibers. Anetoderma is divided into two forms: primary, which is idiopathic and occurs on clinically normal skin, and secondary, which follows a prior dermatosis. Although it is indolent, the lesions of anetoderma persist and may be associated with significant aesthetic changes causing potential psychosocial difficulties. Anetoderma has been successfully treated with ablative, pulsed dye and non-ablative fractionated lasers. Patients with secondary anetoderma and anetoderma limited to a relatively small body surface area may be more amenable to laser treatment than patients with extensive involvement.
Highlights
Primary anetoderma is a rare elastolytic disorder characterized by well-circumscribed flaccid, atrophic macules and patches caused by focal loss of elastic fibers.[1]
A 23 year old woman with no significant past medical history presented to the clinic for evaluation of “spots” that gradually appeared over 2-3 years
Primary anetoderma has historically been subdivided into two subtypes: the Jadassohn-Pellizzari type, with preceding inflammation, and the Schweninger-Buzzi type, which appears spontaneously.[4]
Summary
CASE PRESENTATIONPrimary anetoderma is a rare elastolytic disorder characterized by well-circumscribed flaccid, atrophic macules and patches caused by focal loss of elastic fibers.[1]. Primary anetoderma is a rare elastolytic disorder characterized by well-circumscribed flaccid, atrophic macules and patches caused by focal loss of elastic fibers.[1] Lesions herniate or bulge with palpation or pressure and are described as having a “saclike” appearance.[1] Anetoderma is divided into two forms: primary, which is idiopathic and occurs on clinically normal skin, and secondary, which follows a prior dermatosis.
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