Abstract
B i r t c t f c f i ( M c fl S e m w p c b b i a R 0 d t T e O t S r n t esults: Our data demonstrate that siRNA argeting u-PAR markedly reduced cell roliferation, and increased cell apoptosis, nd suppressed tumour growth, accomanying with the efficient and specific nhibition of endogenous u-PAR expresion in pU group than that in Mock, EV, nd pUc groups. onclusion: These findings suggest that NA interference targeting u-PAR could ffectively inhibit cell proliferation and umour growth of OSCC in vivo. Thus, t may be used as a potent and specific herapy for oral cancer.
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