Abstract
OBJECTIVE: Altered regulation of apoptosis in eutopic endometrium could contribute to survival of endometriotic cell and developed endometriosis. NAG-1, member of transforming growth factor – β (TGF- β) superfamilies, concerns in induction of cell apoptosis. We hypothesized that decreased expression of NAG-1 of eutopic endometrium may be involved in the pathophysiology of endometriosis and investigated the expression of NAG-1 in eutopic endometrium of endometriosis and normal endometrium.
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