Abstract
Background and Aims: Eph receptors (Ephs) and their ephrin ligands are related to a plethora of physiological and pathological processes including atherosclerosis. Preliminary data of our lab showed EphB2 to be the most abundantly expressed Eph receptor on monocytes, one of the culprit cell types in atherosclerosis. Combined with the observation that the EphB2 receptor gene is located on a myocardial infarction locus, we hypothesized that monocytic EphB2 plays a role in atherosclerosis and we investigated the expression and function of EphB2 in human atherosclerosis.
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