Abstract
In nonimmune-modified pancreatectomized total pancrease allograft recipients, heterotopic transplants exhibit elevated plasma insulin values and decreased blood glucose determinations. This is not true of orthotopic allografts. Slight obstruction to venous outflow and concomitant duct ligation have been found to be important in genesis of necrotizing pancreatitis in heterotopic allografts. Rejection of a nonduct-ligated total pancreaticoduodenal allograft is signaled early by hyperamylasemia which appears earlier than the hyperglycemia of islet cell functional shutdown.
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