Abstract
The finding during the nineteen-fifties that carcinogens depress the immune response led to the hypothesis that the rise of malignomas might be correlated with the impairment of the immunocompetent system (1–3). This hypothesis was confirmed by systematic investigations of these relationships by STJERNS-WARD, and especially by the comparison of the activities of carcinogenic and noncarcinogenic hydrocarbons, which revealed that in contrast to noncarcino-genic substances, carcinogens caused immunosuppression (4–6).
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