Abstract
Susceptible dogs were brought into contact with dogs experimentally infected with an avian-origin influenza A virus (H3N2) that had been isolated from a pet dog with severe respiratory syndrome. All the experimentally infected and contact-exposed dogs showed elevated rectal temperatures, virus shedding, seroconversion, and severe necrotizing tracheobronchitis and bronchioalveolitis.
Highlights
Susceptible dogs were brought into contact with dogs experimentally infected with an avian-origin influenza A virus (H3N2) that had been isolated from a pet dog with severe respiratory syndrome
Clinical signs, including sneezing, nasal discharge, and coughing, were observed 2–8 dpi in the challenge group and 5–8 dpi in the exposure group
Twenty-four hours after inoculation, fever developed in dogs in the challenge group that lasted until 3 dpi
Summary
Susceptible dogs were brought into contact with dogs experimentally infected with an avian-origin influenza A virus (H3N2) that had been isolated from a pet dog with severe respiratory syndrome. Transmission of highly pathogenic avian-origin canine influenza A viruses (H3N2) that spread across South Korea during May through December 2007 was observed repeatedly in the country’s animal clinics [1]. The Study Dogs in the study comprised 3 groups of beagles housed in different rooms of the isolation facility at Green Cross Veterinary Products (Yong-in, South Korea). The virus used was avian-origin canine influenza virus A/ canine/01/2007, subtype H3N2, which had been isolated from a pet dog with severe respiratory syndrome.
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