Abstract
B cells contribute to chronic inflammatory diseases via antigen presentation, antibody and cytokine production. Though B cells were associated with increased IgE production in mild allergic asthma and autoantibodies in nasal polyps, their role in severe eosinophilic asthma (EA) is less understood. We characterize the phenotype and function of sputum B cells in 3 groups: healthy controls (HC), patients with eosinophilic asthma on moderate-high dose inhaled corticosteroid (ICS-EA) and oral corticosteroid (OCS-EA).
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